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The presence of diabetes and hyperglycemia tends to shift monocytes towards a functionally proinflammatory phenotype, which induces chronic inflammation and insufficient M2 functional profibrotic TGF- signaling [29] similar to that seen in G6PD-deficient subjects

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Steinbrenner J, Linden H (2001) Regulation of two carotenoid biosynthesis genes coding for phytoene synthase and carotenoid hydroxylase during stress induced astaxanthin formation in the green alga Haematococcus pluvialis

doi: 10.1016/s0140-6736(74)92649-x

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