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In addition, V 0 -ATPase is necessary for amino acids to activate mTOR, thus supporting the view that V 0 -ATPase is an indirect, but integral, component of the mTORC1 pathway (133)
This ATP deficiency disturbs multiple ATP-dependent processes, such as electrolyte transport, substrate utilization, and contractile protein interactions, resulting in impaired myocardial contractility
doi: 10.1016/j.jtcvs.2008.06.023
