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[DOI] [PubMed] [Google Scholar] 237.Kalpage H.A., Bazylianska V., Recanati M.A., Fite A., Liu J., Wan J., Mantena N., Malek M.H., Podgorski I., Heath E.I., et al

doi: 10.1385/BTER:88:1:25 91 MoncayoRKroissAOberwinklerMKarakolcuFStarzingerMKapelariKet al

Exogenously, they arise from exposure to environmental factors like ultraviolet (UV) radiation, pollution, tobacco smoke, and industrial chemicals [3]

recently showed that direct GPx4 overexpression in the cellular models (A549, H460 and H1299 and H23) of non-small cell lung carcinoma (NSCLC) prevented the radiation-induced lipid peroxidation and, in turn, contributed to radioresistance by blocking the ferroptosis mode of cell death [56]

They induce apoptosis in tumor cells via both intrinsic and extrinsic pathways, often associated with the activation of pro-apoptotic proteins and caspase cascades