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Markers of Oxidative Stress and Antioxidant Defense in Romanian Patients with Type 2 Diabetes Mellitus and Obesity
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FAK-paxillin activation for fibroblast migration Growth hormone receptor upregulation on tendons VEGFR2-Akt-eNOS pathway for blood vessel formation Nitric oxide modulation reducing inflammation Erg-1 transcription factor inhibition Angiogenesis, the formation of new blood vessels, represents another critical mechanism
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Nevertheless, one of the immediate ramifications of TBI pertains to preserving the BBB, as the impairment of the BBB following TBI contributes to subsequent harm
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