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Abstract Primary carnitine deficiency (PCD) caused by pathogenic variants in the solute carrier family 22 member 5 ( SLC22A5 ) gene is a rare autosomal recessive disease that results in defective fatty acid oxidation

This metabolicimmune interplay not only reinforces therapeutic resistance but may also contribute to the establishment of a metabolic protective niche, providing a sanctuary within the multicellular tumor ecosystem that shields resistant subpopulations from treatment-induced stress

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An enzyme named methionine synthase is responsible for the conversion of homocysteine back into methionine

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Treatment with carnosine (2 g/day for 8 weeks) and risperidone showed good tolerability and significant beneficial effects on negative symptoms in patients with stable disease [80]