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This suggested that SF was exerting a pharmacodynamic action that reflected induction of GSTs by SF, thereby shunting the reactive epoxide intermediate away from nucleophiles in DNA, where it would have mutagenic and carcinogenic effects, and toward GSH, which allowed it to be further metabolized and excreted in the urine

These processes are influenced, as mentioned, by both exercise and nutrition, which individually and synergistically contribute to neural growth and connectivity

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A comparable approach was previously used by Thomas et al

In contrast, the NP in mice have a highly cellularity, while the human NP have a greater proportion of ECM