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Fast, local rearrangements reflect the entropic contribution

Hepatic lipotoxicity couples peroxisomal -oxidation (H 2 O 2 ) with mitochondrial ROS andwhen labile iron is availabledrives lipid peroxidation and cell-death switching toward ferroptosis, accelerating NASH (Yu and Song 2024)

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The expression level of ET-1 in keratinocytes is increased upon exposure to exogenous insults, and ET-1 promotes the repair of oxidative DNA damage by inhibiting the generation of hydrogen peroxide [61, 62]

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In addition, curcumin supplementation was shown to reduce insulin resistance and improve measures of pancreatic -cell function and glucose tolerance