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These effects were mediated by an increase in phosphorylation of mTOR and/or its downstream substrates, p70-S6 kinase (S6K) and the Eukaryotic translation initiation factor 4E-binding protein-1 (4E-BP1) [21]
doi: 10.1016/S0022-5347(01)64222-8
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10.1016/j.jpainsymman.2008.03.021
doi: 10.1080/15548627.2016.1239003
However, the current largest body of evidence, in the field of metabolic adaptation of cancer cells, deals with the flexible utilization of glutamine and glucose as the main sources of energy
