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dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease Copper homeostasis and cuproptosis in

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ROS level was measured in WI-38 cells treated with 150uM hydrogen peroxide (H 2 O 2 ), with or without pretreatment of GHKCu

dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease Copper homeostasis and cuproptosis in

The presence of a detectable inactive oxidized form of GSTP1-1 has been recently found in salivary samples of healthy human subjects

dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease Copper homeostasis and cuproptosis in

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dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease Copper homeostasis and cuproptosis in

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dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease Copper homeostasis and cuproptosis in

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dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease Copper homeostasis and cuproptosis in

Medical supervision is recommended

dosage of glutathione for copper deficiency improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, and apoptosis induced by deposition in mice with Wilson disease Copper homeostasis and cuproptosis in

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