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Xing J.-F

doi: 10.1093/ajcn/50.2.280

5,7-Dihydroxy-4-methylcoumarin (D4M) markedly regulated p-c-Jun N-terminal kinase (p-JNK) and elevated the expression ratio of p-FoxO1/FoxO1, thus attenuating SGNs injury, mitochondrial dysfunction, ROS accumulation and cisplatin-induced caspase-dependent apoptosis (Li et al., 2023)

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These ligands interact with several receptor proteins including AT 1 , AT 2 , AT 4 , and Mas distributed within the central and peripheral nervous systems as well as local RASs in several organs