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Correspondingly, treatment with -toxin alone had no promoting effect on the production of G-CSF in human umbilical vein endothelial cells (HUVECs), whereas the toxin increased G-CSF production in the presence of PGN (Fig

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Platt, Inhibition of N-glycan processing in B16 melanoma cells results in inactivation of tyrosinase but does not prevent its transport to the melanosome, J

Secondary Carnitine deficiency Secondary deficiency is characterized by increased carnitine excretion in urine in the form of acyl-carnitine due to an accumulation of organic acids [36, 37]