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[DOI] [PubMed] [Google Scholar] 68.Noebels J, et al

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Experiments in animal models have confirmed that high LH levels accelerate the formation of atherosclerotic plaques through a molecular mechanism involving the inhibition of the PI3K/Akt/eNOS signaling pathway
Beginning with the discovery of the part of cysteine-glutathione (GSH)-glutathione peroxidase 4 (GPX4) signaling in inhibition of ferroptosis, the executioner position of lipid peroxidation in ferroptosis is now convinced

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