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Description
However, the inhibition of VLDL uptake in mice overexpressing human apoC1 was also observed in the context of LDL-R deficiency, suggesting that apoC1 inhibition of VLDL uptake involves another pathway than LDL-R, probably LRP [65]
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A lipase fusion feasts on fat
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10.1186/s12864-020-06944-w Summary Keywords TMAO (trimethylamine oxide), MAFLD (metabolic associated fatty liver disease), trimethylamine, TMA precursor, FMO3, unfolded protein response, oxidative stress Citation Li X, Hong J, Wang Y, Pei M, Wang L and Gong Z (2021) Trimethylamine-N-Oxide Pathway: A Potential Target for the Treatment of MAFLD
