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The number of patients who experience increased blood pressure as they progress through the program is relatively small

The above carbon source-dependent phenotypes and physiological alterations directly or indirectly regulate the pathogenicity of C

Appendix Table 1

Sarris AH, Kliche KO, Smith T, Mesina O, Preti A, Hagemeister FB, McLaughlin P, Romaguera J, Rodriguez MA, Pugh W, Fuller L, Ha C, Cox J, Andreeff M , Cabanillas F

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Functional experiments further showed that overexpression of PAX5, one of the genes upregulated in T2D, led to reduced insulin secretion, suppression of mitochondrial respiration, and disruption of energy balance through regulation of downstream genes such as BARX1, NEFL, PCOLCE2, and SFRP1 (Elgamal et al., 2023)