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The N-terminal thiolate in CGHC attacks a substrate's disulfide to form a mixed-disulfide intermediate
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GSTP1 conjugates and detoxifies ethacrynic acid (Ahokas et al, 1984), which was significantly more toxic to GSTP1 null cells (1.3-fold increase in IC 50 , P =0.004, Figure 2A)
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The substance of interest doesnt be able to bind the ligand unless the spacer arm is long enough, but it is also possible to shorten the spacer arm in salt buffer [19]
Pharmacological modulation of the mitochondrial electron transport chain in paclitaxel-induced painful peripheral neuropathy
