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Proc Natl Acad Sci USA 100:54075412

(a) Mice were treated with continuous administration of 15 mM DL -buthionine-(S,R)-sulfoximine (BSO), an inhibitor of glutathione biosynthesis (b), BSO treatment increased the renal mRNA quantity of several genes involved in glutathione biosynthesis, glutathione redox status, and proteostasis, (c) we hypothesize that BSO-induced changes in cellular redox status and gene expression ameliorates proinsulin aggregation and increases the functional insulin levels in -cells, and (d) BSO treatment significantly improved glucose intolerance in AK mice

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Arneth B., Tumor Microenvironment, MedicinaLithuania 56, no