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Mendias has no conflicts of interest that are directly relevant to the content of this article

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Through activation of the Nrf2antioxidant response element (ARE) signaling axis, GSH upregulates cytoprotective enzymes such as heme oxygenase-1, glutathione peroxidase, and NAD(P)H quinone oxidoreductase-1, thereby enhancing cellular resistance to oxidative injury [10]

Intervention studies employing dopaminergic neuron-targeted PGC-1 overexpression via adeno-associated viral vectors reveal enhanced redox homeostasis in striatal neurons and marked attenuation of programmed dopaminergic cell death, establishing this transcriptional coactivators neuroprotective capacity in PD pathophysiology (Wang et al., 2019c)

The isolation window was set to 0.7, and the fixed first mass was 120 m/z

Heidari A., Yazdanpanah N., and Rezaei N., The Role of TollLike Receptors and Neuroinflammation in Parkinson's Disease, J Neuroinflammation 19, no