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The anticancer effect of apigenin in skin cancer has been attributed to its antioxidant properties via modulation of Mitogen-activated protein kinases (MAPK) and Activator protein-1 (AP-1) in the major cell types of the epidermis, keratinocytes (Hwang et al

Reassuringly, the loss of FH was also associated with the activation of an antioxidant response similar to that observed in Fh1-deficient cell lines (Supplementary Fig 5b)

Selenocysteine tRNA is needed to insert selenocysteine into GPX4 13

Peng et al., 2017)

T2-weighted imaging of the early stage implants showed a unique and bright signal that was clearly contrasted with the surrounding muscles

[2] It regulates melanin production, thereby lightening skin tone and reducing hyperpigmentation