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Expression of Claudin1 (CLDN1) has been shown to be increased in both high-grade dysplasia and ulcerative colitis-associated CRC tissue when compared to ulcerative colitis and normal tissue (95)

Silencing of IGF1R exerted effects similar to miR-133a overexpression suggesting that the tumor suppressive function of miR-133a could depend on the regulation of the IGF1R gene (79)
