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Understanding that your symptoms are rooted in real, physiological processeslike the breakdown of the carnitine shuttle and the failure of mitochondrial energy productioncan be incredibly validating

1 2 3 4 5 "New insights concerning the role of carnitine in the regulation of fuel metabolism in skeletal muscle"

Increasing levels of acetyl-CoA through either pathway has the potential to strongly deplete the levels of CoA needed in the mitochondrial matrix for beta oxidation of fatty acids

J Child Neurol 1995;10:S40-4

Registration number of the article: R000025857, UMIN: UMIN000022552