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Moreover, SFN through the formation of oxidative stress can promote autophagy in neurons, probably according to a mechanism that involves the Nrf2 pathway, as shown in Nrf2-KO mice, that instead expressed a lower number of genes related to autophagy [27]

Ferroptosis can be induced by genetically deleting GPX4 or by using small molecules that either covalently inhibit GPX4 (e.g., RSL3) or promote its degradation (e.g., FIN56) [33, 39, 40]

Prostacyclin (prostaglandin I2, PGI2) inhibits platelet adhesion and thrombus formation on subendothelium

While evidence indicates that elevated levels of soluble UA, independent of MSU crystals, directly influence the development and progression of neurological disorders, soluble UA showed robust antioxidative and anti-inflammatory effects [9]

The benzyl alcohol preservative in bacteriostatic water has been linked to serious adverse effects including "gasping syndrome," metabolic acidosis, and neurological complications in this age group

Immunohistochemistry of KIM1, SMA, fibronectin, F4/80, nitrotyrosine, 4HNE, MDA, TfR1, and Gpx4 (used for 3,3-diaminobenzidine [DAB] staining) was examined using the following primary antibodies: polyclonal anti-rabbit KIM1 (1:200 dilution, AF1817