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Antioxidant defense : boosted levels of endogenous glutathione and superoxide dismutase in animal studies
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These inhibitors work by altering the amino acid transport dynamics within key metabolic tissues
Introduction Complex I (NADH-ubiquinone oxidoreductase) is the primary electron entry point in mitochondrial electron transport chain [1, 2] and is absolutely required for glucose-stimulated insulin secretion [3]
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R.SpilkaN.JagannathanK.MorrisP.RomerD.et al
