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Chronic administration of AOD9604 in obese mice and rats leads to significant reductions in body weight gain and increases in fat oxidation, without the adverse effects on insulin sensitivity seen with full-length hGH treatment [3][4]

In this study we show multifaceted mitochondrial aberration in AS brain-derived embryonic NPCs, which exhibit elevated mitochondrial membrane potential (m), lower levels of endogenous reduced glutathione, excessive mitochondrial ROS (mROS) levels, and increased apoptosis compared to wild-type (WT) littermates

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