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For instance, the aforementioned study revealed that inhibiting autophagy via the activation of the class III PI3K pathway during the early stages of cisplatin treatment protected auditory cells, whereas in the later stages, increased autophagy activation through mTOR inhibition led to cell death

These findings underscore the unique pathological timeline and molecular changes in the TgAPParc model, providing a valuable tool for studying Alzheimers disease pathogenesis and therapeutic development

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