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Mechanistically, FSP1 exerts its effects through the quinone oxidoreductase activity associated with NADH, converting ubiquinone to ubiquinol, which in turn inhibits lipid peroxidation and ferroptosis (80)

point out that the peptide DJK-5 showed a strong performance in inhibiting biofilm development and eradicating pre-formed oral biofilms in the short term, compared to L-enantiomeric peptide 1018

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The results of this study suggest a potential role for butyrate both in cardiovascular disease prevention and in therapeutic intervention of arterial restenosis and in-stent restenosis as a pharmacological agent, respectively [27]

doi: 10.1146/annurev.pharmtox.39.1.67