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Keywords Liver disease, Gutliver axis, Gut microbial dysbiosis, Microbial metabolites, Gut barrier, Ecological niche remodeling, Therapeutics, Multi-omics Introduction Liver diseases are one of the leading causes of death and disability worldwide, with a broad spectrum ranging from metabolic dysfunctionassociated steatotic liver disease (MASLD)/nonalcoholic fatty liver disease (NAFLD), alcoholic liver disease (ALD), and viral hepatitis to cirrhosis and hepatocellular carcinoma (HCC)

P-value 1.2 were considered as significantly differentially expressed proteins (DEPs)

Contrarily, the findings of only one experimental eye with less severe circumstances [125,126,127,128,129,130,131] may have more limitations

have constructed mice with cardiomyocyte-restricted deletion of subunit 1 of MPC (cMPC1 / )

Citations Citations to this article as recorded by Glucagon secretion by pancreatic alpha-cells requires an intact tubulin-cytoskeleton-primary cilium axis Elena Casanueva-lvarez, Alba Sanz-Gonzlez, Alicia Vilas, Patricia Cmara-Torres, Magalie A

Simultaneously, under hypoxic or pathological stimuli, dysregulated non-coding RNAs act as key regulators by interfering with Glutathione Peroxidase (GPX4)-mediated antioxidant defense and acyl-CoA synthetase long-chain family member 4 (ACSL4)-mediated lipid metabolism [130, 131], leading to Fe 2+ overload and the accumulation of lipid peroxidation products malondialdehyde (MDA)