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Variances in circadian phase, light responsiveness, and clock gene expression identified between older and younger adults [41], could lead to a different reaction to vitamin B12
Senescent stimuli endow cells with resistance to apoptosis, which involves high activity of anti-apoptotic molecules, such as BCL2 and BCL-XL, and reduced pro-apoptotic BAX protein
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The study from Zheng et al
This response was caused primarily by GH hypersecretion, as expression of a GH antagonist resulted in improvement of glucose homeostasis [129, 229]
