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Ablation of ferroptosis regulator glutathione peroxidase 4 in forebrain neurons promotes cognitive impairment and neurodegeneration

Oxidative stress in polycystic ovary syndrome

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From the perspective of the mitochondrial hypothesis of AD and the possibility of pharmacologically influencing mitochondrial dysfunction, the synaptic plasticity and metabolism of brain cells, CoQ 10 , and mitochondrial proteins and lipids interacting with it also appear to be potential targets for new AD drugs

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[1] Rosen RC, et al