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The therapeutic bioavailability of systemic N-acetylcysteine following TBI is a function of both the regulation of free levels in the blood and permeability of the blood-brain barrier

At the genetic level, it has been observed that the expression of Gln metabolism-related genes in tumor cells is significantly higher than that in CD8+ T cells, underscoring the metabolic advantage held by tumor cells (16)

Efflux inhibition by H 2 S confers sensitivity to doxorubicin-induced cell death in liver cancer cells

In parallel, oxidative stress caused by ROS can be reduced by improving mitochondrial function, which is a major source of ROS in vivo [130,176]

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