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Results Defective in vivo response to eTLRs in Munc13-4 deficiency We previously showed that Munc13-4 binding to STX7 regulates the eTLR response to CpG 16

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[ 433 , 434 ] thalassemia patients suffer from secondary iron overload due to enhanced gastrointestinal iron absorption, [ 435 ] and the accumulation of excessive unmatched globin chains causes premature red blood cells (RBC) hemolysis, which increases iron turnover

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Furthermore, the gut microbiota influences viral clearance and tissue repair by regulating the migration of Th17 cells and group 2 innate lymphoid cells (ILC2s) to the lungs