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APOL-1 alleles APOL-G1 and APOL-G2 specifically damage podocytes and promote kidney injury[116]
Previous studies have shown that, under certain circumstances, such as the induction of ER stress or blockade of ER-to-Golgi transport, the ER core-glycosylated form of CFTR, including the F508 mutant, can be transported to the cell membrane via the UPS pathway, bypassing the Golgi apparatus 12,13,14,15
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This mechanical unloading allows damaged cartilage to recover to some degree and reduces inflammation
Gln is imported via transporters, and its catabolism begins with conversion to glutamate
On the function of the NADP-dependent isocitrate dehydrogenase isoenzymes in living organisms
