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Nevertheless, the actions of the IGF-1 are attenuated in the context of renal disease, which may be promoted by the presence of inhibitory factors, such as the IGF binding proteins, and inability to trigger intracellular downstream signaling despite of normal IGF-1 receptor expression in cell surface

Ferroptosis and nanotherapy Despite the remarkable antitumor potential of ferroptosis targeted therapy observed in preclinical settings, its clinical translation is impeded by intrinsic limitations: the majority of ferroptosis regulators exhibit inadequate biocompatibility, poor aqueous solubility, and insufficient targeting ability, rendering them prone to causing off target effects

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doi: 10.1074/jbc.M114.559518 115 BrayFFerlayJSoerjomataramISiegelRLTorreLAJemalA