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Lanosterol has been found to inhibit transmembrane protein 175 (TMEM175), a lysosomal proton export channel, and its clearance via CYP51A1-dependent metabolism restores TMEM175 function, thereby dampening lysosomal alkalinization and mitigating the induction of alkaliptosis [68]

Taking both together improves the overall effect

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Her dystonia remained controlled with regular botulinum toxin injection and low dose levodopa, anticholinergic, and benzodiazepine