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Increased flux of key cellular energy regulators, including NAD + and SAM, may potentially define the therapeutic mechanism-of-action of NNMT inhibitors

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In PEG-repaired animals, the aberrant label found beyond the mediolateral boundaries defined in Unoperated Control animals may be explained by PEG-induced misconnections between distal sensory axons projecting from the dorsal aspect of the foot via the peroneal branch being fused to inappropriate proximal axons of the tibial and sural branches

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