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Notably, the -glutamyl peptide analogues of cysteine and GSH such as 2-aminobutyric acid (2AB) and ophthalmic acid (OA or -glutamyl-2-aminobutyryl-glycine) were increased in the serum of cysteine-deficient mice (Fig

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This effect may result from T-cell hyporesponsiveness induced by continued ROS exposure, which was induced by imbalance between ROS generation and scavenging through inhibition of antioxidant activity of Gpx1