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In addition to effects on lipid metabolism, apoC-III has been shown to directly influence development of atherosclerosis by several routes including facilitating subendothelial accumulation of atherogenic lipoproteins by increasing their affinity for artery wall proteoglycans [21,22,23,24,25,26]
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Conversely, AAV-mediated inhibition of miR-128 expression was found to attenuate METH-induced addictive behavior
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