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Epidemiological, clinical, and experimental studies have comprehensively demonstrated that both the varieties and amounts of dietary protein intake, as well as AA supplementation, can affect the pathogenesis of intestinal inflammation in various ways [131]

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[45] recently found that peroxisomes induce ferroptosis by synthesizing polyunsaturated ether phospholipids (PUFA-ePLs), which are substrates of lipid peroxidation
