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These acylcarnitines can be exported from the mitochondria and cell, preventing the toxic accumulation of acyl-CoA intermediates that inhibit key metabolic enzymes

Hemodial Int 15:S68S71 Acknowledgments We would like to thank Dr Chris Chan for reviewing the manuscript and our patient for giving his consent to this publication

Cheng Y, Yan G, Li C, Han X, Shang J, Shang S, et al

This creates a common source of confusion that requires careful attention

Additional support for a causal role of mitochondrial dysfunction in neuroinflammation comes from lipopolysaccharide (LPS)-induced depression models