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Mitochondrial dysfunction in sepsis: mechanisms and therapeutic perspectives
89 ROS may also activate antiangiogenic pathways, such as p53-mediated production of thrombospondin-1
D-serine has been known as physiological functions, such as CNS development, and pathology, such as neuro-psychiatric and neurodegenerative diseases related to NMDA receptors dysfunction (Yamamori et al., 2014
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Oxid Med Cell Longev 2013:110 Moreira PI, Carvalho C, Zhu X, Smith MA, Perry G (2010) Mitochondrial dysfunction is a trigger of Alzheimers disease pathophysiology
