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In this context, the development of new primary melanomas on pre-existing nevi (12%), detected by clinical and microscopic changes at approximately 14 weeks, has also been described histopathologically [92]

These CRP monomers, predominantly located in tissue, may enhance inflammatory mechanisms, favouring the transendothelial migration of leukocytes, monocyte activation through Fc-RI/III receptors and the activation of complement inhibitors factor H and C4b-BP.58 The neutral results with pCRP infusion coincide with the data obtained from Mendelian randomisation studies which confirm that CRP is a predictor of cardiovascular events but is unlikely to play a direct aetiopathogenic role in the inflammatory processes associated with atherosclerosis, in contrast to the powerful evidence for IL-6 or IL-1, which also seem to have a causative role in the pro-inflammatory process.59,60 Hs-CRP may be the surrogate biomarker for the activation of this pro-inflammatory pathway mediated by IL-1 and IL-6

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