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Description
Developing combinational therapies that address neuronal hyperexcitability as well as oxidative stress may prevent the deleterious cycle of neuroinflammation and oxidative stress that contribute to epileptogenesis
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doi: 10.1186/s12906-018-2191-z 85 DehghaniSAlipoorESalimzadehAYaseriMHosseiniMFeinle-BissetCet al
Clin Neuropharmacol 2013;36, 103-106
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Matrix Regulation and Remodeling Laboratory observations indicate that GHK-Cu may play a part in modulating metalloproteinases (MMPs) along with their inhibitors (TIMPs)
