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The disbalance between the inhibited enterocytic influx and unchanged or increased systemic demand may cause specific vulnerability of the gut epithelium to toxic effects of the thiamine-competitive drugs targeting SLC19A3

doi:10.1016/j.jplph.2007.10.015

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Therefore, the growth disparity in 3D gels may stem from a distinct mechanism

Role of TLR4/NADPH Oxidase 4 Pathway in Promoting Cell Death through Autophagy and Ferroptosis during Heart Failure

S100 chemokines mediate bookmarking of premetastatic niches