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The pathophysiology of synaptic loss and neurodegeneration involves problems in the cleavage of the amyloid precursor protein (APP) with the production of APP beta-amyloid (A) fragment, together with the aggregation of the hyperphosphorylated tau protein

Reactive oxygen species production by monoamine oxidases in intact cells

Strikingly, selective deletion of EAAT2 in spinal cord astrocytes is sufficient to induce motor neuron degeneration and impairs motor learning and coordination in mice [391]

Despite this, current evidence underscores that the behavioral gains are generally not permanent without ongoing supplementation

Other studies have shown that selenium stimulates the expression of the pancreatic beta cell gene and improves islet function in cell cultures, suggesting its antidiabetic potential due to its insulin-like properties

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