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Overexpression of LIAS reduced hepatic oxidative stress in non-alcoholic fatty liver disease in mice and protected mitochondrial function by upregulating the nuclear factor erythroid 2related factor 2 to reduce ROS production (52), attenuated the chronic inflammatory response, inhibited NF-kB activity in lung fibrosis in mice (53), significantly increased Treg cell numbers, and reduced T cell infiltration (50)

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What the Human Evidence Actually Shows Sadly, we currently dont have massive Phase 3 trials comparing GHK-Cu head-to-head with Finasteride in 5,000 men