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However, a postmortem study showed that GSH and GSH/GSSG were significantly decreased in the brains of ASD patients relative to controls 18 , suggesting that markers of glutathione metabolism may have parallel changes between central and peripheral in ASD

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GGT activity (even when in the normal range) exerts pro-oxidant activity and catalyzes the oxidation of low-density lipoprotein (LDL) within atherosclerotic plaque, promoting atherosclerosis progression (build-up) and plaque instability [16,67]

Diabetic long-term complications are associated with hyperglycemia caused by increased production of reactive oxygen species and attenuated scavenging enzymes [44, 45]

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Authors contributions AKdiagnosed the clinical scenario, managed the patient, manuscript preparation