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During thyroid hormone synthesis, hydrogen peroxide (HO) is continuously generated as part of normal physiology

These results reflect measurable metabolic changes across multiple endpoints

Three mechanistic explanations, not mutually exclusive: Signalling cascade persistence: BPC-157 activates VEGFR2 and FAK-paxillin pathways whose downstream effects (angiogenesis, fibroblast migration, collagen remodelling) unfold over days to weeks after initial receptor engagement

In sequences containing Asp-Pro, the cyclic imide formation can actually cleave the peptide backbone entirely, splitting the peptide into two fragments

Residual solvent / counter-ion Provided when available or required for procurement review

Instead, the size of the alkyl substituents on the positively charged nitrogen seems to be a decisive factor