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Concurrently, a spectrum of inflammatory mediatorsincluding cytokines, chemokines, and proteasesis released, collectively establishing a persistent pro-inflammatory microenvironment that sustains disease progression ( In recent years, targeted studies have further unraveled the complex regulatory mechanisms of chronic inflammation in COPD, spanning molecular pathways, cellular effects, animal models, microecological balance, and immune defense

Oxidative stress and docosahexaenoic acid injury lead to increased necroptosis and ferroptosis in retinal pigment epithelium

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Utilize suitable contamination-control procedures