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These effects (i.e., rapidly reestablished reorganized blood flow) attenuated/eliminated the lesions in the brain (intracerebral and intraventricular hemorrhage), heart (congestion, myocardial infarction, severe arrhythmias), lung (hemorrhage), and congestion in the liver, kidney, and gastrointestinal tract [52,53]

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The activation and proliferation of glial cells, in turn, are useful in order to release signaling factors and to trigger a robust immune reaction consisting of brain-resident as well as peripherally recruited inflammatory cells

This suggests that the loss of membrane integrity and the externalization of PS, which are key features of apoptotic cells, are at least partially driven by caspase activity in mature RBCs

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As PSR-1 is required for ferroptosis signaling-induced axonal fusion, we next tested if PSR-1 condensates were affected by oxidative environment