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Glutamine synthesis is a highly active metabolic pathway in astrocytes, and several distinct metabolic features of astrocytes are required to sustain the extensive synthesis and release of glutamine (highlighted in Figure 1)

As immunotherapeutic sensitivity increases (from nonresponder to pCR), apCAFs co-localized with macrophages (mostly SELENOP-macrophages), accumulated in tumor boundaries and infiltrated within tumor regions, indicating the cooperative roles of apCAFs and SELENOP-macrophages in the anti-tumor activity induced by immunotherapy

amyloid plaques and A immuno-staining, a therapeutic target for AD in APP/PS1 transgenic mice

EGCG inhibits protein synthesis, lipogenesis, and cell cycle progression through activation of AMPK in p53 positive and negative human hepatoma cells

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o In a recent investor call, EVP of R&D Martin Lange highlighted that this higher dose can achieve a full 21% weight loss