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Because of 1 H-MRS results supporting glutamate reductions secondary to antipsychotics, agents developed to increase glutamate, like NMDA agonists, would be unlikely to have a therapeutic role (perhaps beyond correcting some side-effects of D 2 blockers)

As a consequence of cell death, its intracellular components are released as pro-inflammatory damage-associated molecular patterns (DAMPs) such as ATP, nucleotides, histones, and high-mobility group protein B1 (HMGB1) and pro-inflammatory cytokines, such as interleukin1 (IL1), IL33, and interferon (IFN) upon plasma membrane rupture, which triggers the innate immune system [48]

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This provides a means to ensure effective nitrogen trafficking within the mitochondria

doi:10.1007/s12010-011-9436-3 Chen J, Xie L, Cai J, Yang C, Duan X (2013) Enzymatic synthesis of glutathione using engineered Saccharomyces cerevisiae

Ascorbateglutathione pathway and stress tolerance in plants